Print ISSN: 1683-3589

Online ISSN: 2409-501X

Keywords : Anesthesia


MANAGEMENT OF CRISES DURING ANESTHESIA AND SURGERY. PART XI: PULMONARY EDEMA & WATER INTOXICATION

Salam N Asfar; Jasim M Salman

Basrah Journal of Surgery, 2016, Volume 22, Issue 2, Pages 105-108
DOI: 10.33762/bsurg.2016.116623

PULMONARY OEDEMA/ARDS ulmonary edema is a potential cause of hypoxia in the perioperative patient. The accumulation of excessive alveolar fluid results in hypoxia due to interference with diffusion across the alveolar capillary membrane. Frothy (sometimes blood-stained) sputum may be expectorated or observed in the endotracheal tube. the abnormal accumulation of fluid in the interstitial or alveolar spaces of the lung can be explained on the basis of a disturbance in the normal Starling equation1. It involves changes in hydrostatic or oncotic pressure across the alveolar membrane or in the permeability of the alveolar membrane such that fluid moves across from the capillaries into the alveolar space. P

ANESTHESIA USING LARYNGEAL MASK AIRWAY FOR INTRA-NASAL SURGERY; A COMPARATIVE STUDY

Basrah Journal of Surgery, 2005, Volume 11, Issue 1, Pages 43-49
DOI: 10.33762/bsurg.2005.55413

The purpose of the study was to compare the suitability and safety of the laryngeal mask airway (LMA), for intranasal surgery (INS) anesthesia, with endotracheal tube (ET) anesthesia.
we studied 65 patients (ASA grade I and II, according to American Society of Anesthesia classification), aged (18-39) years. The study population was scheduled for elective intranasal surgery. The patients were randomly assigned into two groups: the first group (33 patients), a laryngeal mask airway (LMA Group) was inserted under propofol , fentanyl and muscle relaxant (atracrium), anesthesia was maintained by using a mixture of halothane in N2O/O2. The second Group (32 patients), an endotracheal tube (ET Group) was inserted under propofol, fentanyl and muscle relaxant (atracrium), anesthesia was maintained with a mixture of halothane in N2O/O2.
All complications concerning airway insertion, removal or interruption of surgery for compromised airway and ventilation were recorded. Mean blood pressure, heart rate and pulse oxymetry, were continuously monitored and recorded before and after induction and airway device insertion, followed by 10 minutes intervals. Data were analyzed using chi square statistical test; Null hypothesis was rejected at P> 0.05.
In LMA Group, there were no episodes of post removal laryngospasm. The incidence of oxyhemoglobine desaturation at removal was significantly reduced compared with that in ET Group (P< O.O2). The number of patients with oxyhemoglobine desaturation less than 92% on airway device removal was 0% in LMA Group, 3 patients (9.375%) in ET Group.
In ET Group, the mean blood pressure and heart rate showed significant variation between the different time measurements (P> 0.005). Intubation and extubation resulted in significant transient increase in mean blood pressure and heart rate. In LMA Group, the mean blood pressure was less than baseline value from 1 minute after induction onwards (P< 0.005) and did not show any significant changes during the different time points measurements. LMA application or removal did not cause any significant increase in mean blood pressure or heart rate (P< 0.001).
We conclude that using LMA is suitable method for intranasal surgery. It provides a safe, protected airway with a smoother emergence from anesthesia than tracheal intubations. Anesthesia using LMA for intranasal surgery provides a stable circulation.